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  The Itaconate Shunt Hypothesis, proposed by Dr. Robert Phair in collaboration with Dr. Ron Davis and supported by the Open Medicine Foundation, suggests a novel metabolic mechanism that may underlie the persistent symptoms of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS). Normally, the TCA (Krebs) cycle in mitochondria efficiently produces energy using glucose and fatty acids.